May 17, 2024
Phlegmonous Gastritis
Emphysematous Gastritis
H.Pylori Gastritis
Lymphocytic Gastritis
Eosinophilic Gastritis
AMAG / EMAG against Type B Gastritis. Selective forms of gastritis.
The most popular: NSAIDs Treatment: supportive (PPI/H2 blocker) Viruses > Bacteria > Fungi are the second most common causes.
Alternatively known as suppurative gastritis; causative factors The most prevalent organisms are streptococci or E. coli. Patients with chronic debility or immunocompromised individuals are usually affected by an uncommon form of gastritis.
Features in clinical context
• Severe fever or pain in the abdomen
• The infection affects the muscle level, or lamina propria; the imaging may demonstrate the thickened stomach wall. Mucosa plays no role.
• connected to gas or pus-filled pockets; thicker stomach walls
Treatment options include: gastroplasty, early broad-spectrum treatment, antibiotics, vanco and piptaz.
Bacteria that produce gas, such as S. aureus, E. coli, and Cl. perfringens The death rate exceeds forty percent. There are gas pockets visible in the stomach wall.
Treatment options include: gastroplasty, early broad-spectrum treatment, antibiotics, vanco and piptaz
Gastritis caused by the pylori virus; Chronic Atrophic Gastritis; Differential Forms; Collagenous; Lymphocytic/varioliform; Eosinophilic; Gastritis cystica profunda
Features Type A Type B Etiopath Autoimmune H. Pylori Patient Characteristic HLA B8/D3 - Gastric secretion Decrease Increase Distribution Fundus Antrum Pernicious Anemia Common Uncommon Gastrin levels Antral sparing
AMAG → AMAG/EMAG OVERLAP → EMAG Antibodies to intrinsic factor, parietal cell HP gastritis (Current, past) Other auto-immune disorders Potentially reversible (Hp, Rx) Antral Sparing Antral involvement Serum PGI and PGI / PGII ratio Serum PG levels are more variable Hypergastrinemia (can be marked) Normal or slight increase in serum gastrin Gastric carcinoid tumors
OLGA and OLGIM Classifications of Cancer Risk in Chronic Gastritis | |||||
OLGA staging | |||||
Corpus (Body, Fundus) | |||||
Atrophy | None (0) | Mid (1) | Moderate (2) | Severe (3) | |
Antrum | None (0) | Stage 0 | Stage I | Stage II | Stage II |
Mid (1) | Stage I | Stage I | Stage II | Stage III | |
Moderate (2) | Stage II | Stage II | Stage III | Stage IV | |
Severe (3) | Stage III | Stage III | Stage IV | Stage IV | |
OLGIM Staging | |||||
Corpus (Body, Fundus) | |||||
Antrum | Intestinal Metaplasia (IM) | None (0) | Mid (1) | Moderate (2) | Severe (3) |
None (0) | Stage 0 | Stage I | Stage II | Stage II | |
Mid (1) | Stage I | Stage I | Stage II | Stage III | |
Moderate (2) | Stage II | Stage II | Stage III | Stage IV | |
Severe (3) | Stage III | Stage III | Stage IV | Stage IV |
Collagenous gastritis: adults get diarrhea, and children experience abdominal pain. A condition in which the lamina subepithelial area exhibits enhanced collagen deposition Biopsy reveals normal modules; endoscopy demonstrates mucosal modularity.
There are more than 20 microns of collagen.
OGD, or varioliform gastritis; • H pylori and celiac disease; • nodule formation and central atrophy.
• IEL in the propria lamina
More than 20 Eosinophilic per hpf; more than 50% of patients have peripheral eosinophilia; antral region has greater edema; the patient may present with recurrent food infections in the esophagus; an obstructive presentation may arise.
Also Read: Recent Studies And Trials In Gastroenterology And Hepatology
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